As far as I’m concerned, the final nail in the genetic-testing coffin was struck when GSK (GlaxcoSmithKline) announced “GSK Buys $300 Million Stake in 23andMe for Drug Research Data.” Yes, that’s right, GSK is buying into 23andMe so it can collect your data to develop new drugs and identify if you should be entered into a particular drug trial.
The Pollyanna part of Alternative Medicine looks at genetic testing as a way for people to learn what potential genetic problems can be headed off with lifestyle changes. But I really don’t think that is a realistic assumption. If someone finds out they have the gene for Alzheimer’s and allopathic medicine says they have a drug for it – the patient is most likely going to choose the drug. It won’t be long before your 23andMe print out will come with a list of drugs you can take.
Here’s how GSK is spinning this partnership “By leveraging the genetic and phenotypic information provided by consenting 23andMe customers and combining it with GSK’s incredible expertise and resources in drug discovery, we believe we can more quickly make treating and curing diseases a reality.”
Of course you should be asking how effective has any drug company been in curing any disease with their “incredible expertise and resources”. These people aren’t even smart enough to know that 80% of their “customers” are magnesium-deficient which changes the whole playing field.
What are they working on first? Parkinson’s Disease. They are recruiting patients to help test a new GKS treatment for Parkinson’s. As is usually the case, the drug inhibits something. They want to inhibit the “leucine-rich repeat kinase 2 (LRRK2) gene, which encodes the protein dardarin.” So if you have a variant of the LRRK2 gene you will be notified that you are eligible for a clinical trial. Maybe the drug will help and maybe it won’t but why aren’t you told that Parkinson’s could be something entirely different. Here is an excerpt from The Magnesium Miracle (2017) about Parkinson’s.
Unfortunately money is not being put into human research on magnesium and Parkinson’s but there has been a flurry of animal research. In the 2015 review, “Magnesium in Man: Implications for Health and Disease” the authors state, “Parkinson’s patients have low Mg2 concentrations in cortex, white matter, basal ganglia, and brain stem. According to the writer “…rats with chronically low Mg2 intake exhibit a significant loss of dopaminergic neurons.” They also showed that “In this experimental model, mitochondrial Mg2 concentrations were decreased.” Several other significant findings allowed them to conclude, “Mg2 supplementation may be beneficial for patients suffering from Parkinson’s disease.”
As with Alzheimer’s, aluminum can be a contributing factor in Parkinson’s. In one autopsy study, calcium and aluminum were elevated in the brains of victims of Parkinson’s disease as compared to people with normal brains. When I hear that excess calcium is involved, as in this study, I know magnesium therapy should be considered. In another study, magnesium was lower in the brain cortex than in the white matter of Parkinson’s brains.
Research indicates that ample magnesium can protect brain cells from the damaging effects of aluminum, beryllium, cadmium, lead, mercury, and nickel. We also know that low levels of brain magnesium contribute to the deposition of heavy metals in the brain that heralds Parkinson’s and Alzheimer’s. It appears that the metals compete with magnesium for entry into the brain cells. If magnesium is low in the brain, heavy metals gain access much more readily.
Of course, I’m not saying that magnesium will “cure” Parkinson’s but it should be part of the treatment protocol. Or better yet, if people are properly saturated with magnesium, they might not develop these neurological diseases. My advice is to keep taking your ReMag and other Completement Formulas to offset the imbalances that could occur without them!
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Carolyn Dean MD ND
The Doctor of the Future®
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