What do you think of when you read the Medscape headline ”High Rate of Bone Deterioration Among Women With T2DM?”
Here are two hints. Magnesium deficiency is a sign of diabetes and osteoporosis is related to magnesium deficiency. So, it’s no wonder that 2/3 of women with type 2 diabetes mellitus have low bone mass and osteopenia. So-called smart doctors with decades of experience in clinical research say that “T2DM increases a person’s likelihood of osteoporotic fracture, boosting the risk by up to 30%, but the mechanism involved remains unclear.”
Did you catch that “…the mechanism involved remains unclear.” LISTEN TO ME! The mechanism is MAGNESIUM DEFICIENCY.
Below is an edited excerpt on magnesium deficiency and osteoporosis from Chapter 11 of The Magnesium Miracle (2017).
OSTEOPOROSIS MISUNDERSTOOD AND MISTREATED
A 2014 meta-analysis confirmed the association between Serum Magnesium levels and postmenopausal osteoporosis. Seven studies involving 1,349 postmenopausal women were identified. Overall, postmenopausal osteoporotic women had lower serum levels of magnesium than the healthy controls.
With drug companies funding most of the osteoporosis research, there are no large clinical trials investigating the magnesium connection in bone production and there probably never will be. As long as people are given false hope that there is some magic bullet in the pharmaceutical pipeline that will “cure” osteoporosis, or any other chronic disease, they will ignore the underlying diet- and nutrient-related reasons for their health problems.
Reports that Fosamax causes jawbone deterioration is evidence that this osteoporosis drug, and likely all bisphosphonates, cause brittle bones. This side effect of jawbone deterioration has dentists refusing to place dental implants in women who are on Fosamax.
Fosamax works by destroying osteoclasts, the cells that break down bone as it ages. These cells also sculpt the bone as new bone forms, shaping it into a strong and stable matrix. Fosamax prevents bone from breaking down—but the drug companies did not reckon with the necessary bone-remodeling function of the osteoclast. Without osteoclasts bones have no blueprint to follow and calcium is deposited helter-skelter. X-rays of bones under the influence of Fosamax may appear dense at a glance, but when you look closely, without the remodeling capacity of osteoclasts, the bones’ internal structure is in complete disarray. These bones are brittle and they break more easily.
Instead of cozying up to Fosamax, doctors should read a 2013 study in the Journal of Nutritional Biochemistry showing that magnesium has a direct influence on increasing the formation of osteoclasts. Investigators acknowledged that magnesium deficiency occurs frequently and leads to loss of bone mass, abnormal bone growth and skeletal weakness. But they wanted to determine how this occurs. Using an animal model, they found that magnesium deficiency inhibited the activity of osteoclasts – which is the mechanism of action of Fosamax. They concluded that altered osteoclast numbers and activity may contribute to the bone changes that are seen in magnesium-deficient patients.
The unfortunate consequence of this study about bones deteriorating in diabetes is that diabetics will be offered high dose calcium and Fosamax, instead of magnesium, to “save their bones!”
Once again doctors come up against evidence of magnesium deficiency in diabetes and make a complete U turn away from prescribing magnesium. But you can see it and you know it makes sense and it’s up to you to take care of your own bones! You can read my ReMyte/ReCalcia book for more.
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Carolyn Dean MD ND
The Doctor of the Future®
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